This research investigates the role of oligodendrocytes in Fragile X syndrome, revealing delayed cell maturation and disrupted myelin development. Using human stem cell and mouse models, it identifies these overlooked cells as promising therapeutic targets, potentially enabling earlier and more effective treatments for Fragile X and related neurodevelopmental disorders.

This research investigates whether zinc plays a critical role in the ability of psychedelic drugs to reopen social reward critical periods in the brain. Using mouse models, the study examines how zinc influences social behavior following psychedelic treatment, potentially revealing mechanisms of brain plasticity relevant to autism, social anxiety, and social connection.